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Patient handout

Acute HF — Type-1 cardiorenal syndrome (ADHF + AKI)

PRODUCTION

1. Your condition

This handout is for acute hf — type-1 cardiorenal syndrome (adhf + aki). Your care team identified this based on: cr rise ≥0.3 mg/dl or ≥50% from baseline during adhf treatment → type-1 crs (ronco 2008; kdigo aki).

Other reasons your team may use this plan: adhf + oliguria <0.5 ml/kg/h despite adequate diuretic dose → cardiorenal phenotype; persistent congestion + escalating loop diuretic + worsening renal function → sequential blockade indication.

2. Your medications

Take these medications exactly as prescribed. Do not stop or change a dose without talking to your provider.

MedicationStarting doseHowWhenWhat it does
furosemideIV bolus 2-2.5× home dose; if naïve 80 mg IV, then 80-160 mg IV q6-12h or continuous 10-20 mg/hIVq6-12h or continuousDOSE PMID 21366472 — high-dose IV bolus or continuous infusion equivalent; assess UOP at 2h; do NOT default to UF per CARRESS-HF PMID 23131078
acetazolamide500 mg IV daily × 3 daysIVdaily × 3ADVOR PMID 36027564 — adjunct to IV loop improves decongestion at 3 days without worsening renal function
hydrochlorothiazide25-50 mg PO dailyPOdailyCLOROTIC PMID 36461706 — HCTZ + IV loop improves decongestion vs loop alone; sequential nephron blockade
metolazone2.5-10 mg PO 30 min before loopPOdaily or BIDSequential nephron blockade alternative; longer half-life than HCTZ; monitor K + Mg closely
empagliflozin10 mg PO daily — CONTINUE if eGFR >20POdailyEMPULSE PMID 35347356 — reno-cardiac protective; continue in CRS if eGFR >20 unless DKA risk; do NOT discontinue for moderate Cr rise
sacubitril-valsartanHOLD if Cr rise >50% from baseline; restart at 24/26 BID at 50% lower dose once Cr stablePOBIDHold during severe Cr rise; restart at lower dose once euvolemic — preserves long-term cardiorenal benefit
enalaprilHOLD if Cr rise >50% from baseline; restart at 2.5 mg BID once Cr stablePOBIDSame hold-then-restart approach; ACEi/ARB withdrawal during severe AKI is appropriate
spironolactone12.5 mg PO daily once Cr stable + K <5POdailyRALES PMID 10471456 — start cautiously after CRS resolution; lower starting dose given K + Cr risk
carvedilolCONTINUE home dose if hemodynamically tolerant; do not reduce solely for AKIPOBIDCOPERNICUS PMID 11386262 — chronic BB withdrawal during ADHF associated with worse outcomes; continue unless hypotensive or bradycardic
dobutamine2.5 µg/kg/min IV titrateIVcontinuousInotropic support to enable diuresis when low CO drives renal hypoperfusion; arrhythmia risk

Plan: Type-1 CRS — sequential nephron blockade to achieve euvolemia while tolerating ≤30% Cr rise (CARRESS-HF; ADVOR; CLOROTIC)

3. When to call your provider

Contact your care team if any of the following happen:

  • Recurrent ADHF → readmit + reassess
  • eGFR decline >5/yr → nephrology + the four foundational heart-failure medications review
  • EF declining → advanced HF eval

4. When to seek emergency care

Call 911 or go to the nearest emergency room right away if you have:

  • Persistent congestion + AKI despite IV loop + acetazolamide + HCTZ/metolazone × 24-48h
  • KDIGO Stage 3 AKI (Cr rise ≥3× baseline OR Cr ≥4 OR UOP <0.3 mL/kg/h × 24h) during ADHF treatment(life-threatening)
  • CRS + SBP <90 + lactate ≥2 + cool extremities + AKI = SCAI C+ shock with renal hypoperfusion(life-threatening)
  • K >6.0 with EKG changes during CRS treatment (sequential blockade + ACEi/ARNI + MRA exposure)(life-threatening)

5. Follow-up

Restart ACEi/ARB or ARNI at lower dose once euvolemic + Cr stable; STRONG-HF cadence; nephrology clinic if AKI did not fully resolve; SGLT2i continuation

6. Sources

Guideline: 2022 ACC/AHA HF + 2023 Focused Update + KDIGO AKI 2026 draft

  1. pubmed.ncbi.nlm.nih.gov/35363499
  2. pubmed.ncbi.nlm.nih.gov/38264914
  3. pubmed.ncbi.nlm.nih.gov/23131078