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Patient handout

Hypertrophic cardiomyopathy with acute decompensation

PRODUCTION

1. Your condition

This handout is for hypertrophic cardiomyopathy with acute decompensation. Your care team identified this based on: acute dyspnea / orthopnea / pre-syncope in patient with known hcm (resting or provocable lvot gradient ≥30 mmhg) — sudden hemodynamic deterioration.

Other reasons your team may use this plan: bedside echo: new or worsened sam-mediated lvot obstruction with dynamic gradient (rest ≥50 mmhg or with valsalva/exercise provocation) in hcm patient; new-onset afib with rapid ventricular response in hcm — loss of atrial kick → catastrophic lv filling drop because stiff hypertrophied lv depends on atrial systole; hcm patient with precipitant: gi losses, sepsis, fever, postoperative volume shifts, or inadvertent diuretic over-shoot → acute lvot obstruction crisis.

2. Your medications

Take these medications exactly as prescribed. Do not stop or change a dose without talking to your provider.

MedicationStarting doseHowWhenWhat it does
phenylephrine50-200 mcg IV bolus or 0.5-3 mcg/kg/min infusion titrate to MAP ≥65IVbolus or continuous infusionPure α-agonist increases afterload → reduces LVOT gradient by improving aortic ejection and abolishing SAM; preferred over norepinephrine (β-component worsens obstruction) and ABSOLUTELY preferred over inotropes
esmolol500 mcg/kg IV bolus then 50-300 mcg/kg/min infusion titrate to HR <80IVcontinuous infusionShort-acting cardioselective β-blocker lengthens diastolic filling time, reduces inotropy → reduces LVOT gradient; titratable, off rapidly if hemodynamic compromise
metoprolol_tartrate25 mg PO BID titrate to maximally toleratedPOBIDACC/AHA 2024 HCM Class I — first-line chronic β-blockade for symptomatic HCM; reduces inotropy + lengthens diastole
verapamil120 mg PO daily extended-release titrate to 480 mg/d maxPOdaily ERACC/AHA 2024 HCM Class I alternative — non-DHP CCB; CAUTION in obstructive HCM with very high gradient or heart failure (vasodilator effect can worsen gradient — start low)
disopyramide40-50 mg PO TID-QID extended-release titrate to 600-800 mg/d max; renal-adjustedPOTID-QIDACC/AHA 2024 HCM Class IIa — negative inotrope reduces SAM and LVOT gradient; QT prolongation + anticholinergic effects + proarrhythmic — monitor QT, K, Mg
mavacamten5 mg PO daily start; titrate q4w per echo per REMS (max 15 mg/d)POdailyACC/AHA 2024 HCM Class IIa — first-in-class cardiac myosin inhibitor; EXPLORER-HCM PMID 32861276 (Olivotto Lancet 2020): 30% relative improvement in pVO2 + ~47% reduction in resting LVOT gradient; VALOR-HCM 2023: reduces septal reduction therapy referrals; REMS program — echo q4w during titration to detect LVEF drop <50%
apixaban5 mg PO BID (2.5 mg BID per FDA dose-reduction criteria)POBID indefiniteACC/AHA 2024 HCM Class I — ANY AF in HCM = anticoagulate regardless of CHA2DS2-VASc score; DOAC preferred over warfarin; RE-LY/ARISTOTLE/ROCKET-AF/ENGAGE class evidence applies
rivaroxaban20 mg PO daily with food (15 mg if CrCl 15-50)POdaily indefiniteACC/AHA 2024 HCM Class I — DOAC alternative for AF in HCM
warfarin5 mg PO daily; INR target 2-3POdaily indefiniteACC/AHA 2024 HCM Class I if DOAC contraindicated; INR 2-3

Plan: HCM acute decompensation — preload/afterload-dependent: fluids + phenylephrine + esmolol + AVOID inotropes/vasodilators; chronic: BB/CCB/disopyramide → mavacamten → septal reduction therapy (ACC/AHA 2024 HCM)

3. When to call your provider

Contact your care team if any of the following happen:

  • Symptomatic progression → escalate medical therapy or septal reduction therapy
  • New VT/VF or syncope → urgent EP / ICD evaluation
  • New AF → AC initiation
  • End-stage dilated phenotype (heart pumping strength (LVEF) <50%) → switch to cardio.hfref.core.v1 for the four foundational heart-failure medications 4-pillar

4. When to seek emergency care

Call 911 or go to the nearest emergency room right away if you have:

  • HCM patient given inadvertent inotrope (dobutamine, milrinone, epinephrine drip) for hypotension — paradoxically worsens SAM-mediated LVOT obstruction with hemodynamic collapse(life-threatening)
  • New-onset AFib with rapid ventricular response in HCM patient — loss of atrial kick + tachycardia → catastrophic drop in LV filling because stiff hypertrophied LV depends on atrial systole(life-threatening)
  • HCM patient with persistent NYHA III-IV symptoms + LVOT gradient ≥50 mmHg at rest or with provocation despite maximally tolerated medical therapy (BB or CCB ± disopyramide ± mavacamten) — septal reduction therapy referral
  • HCM patient on mavacamten with REMS echo showing heart pumping strength (LVEF) drop <50% during titration — REMS-mandated dose reduction or discontinuation
  • HCM patient with unexplained syncope, NSVT on Holter, family history of SCD, LV thickness ≥30 mm, apical aneurysm on CMR, or high LGE burden (>15% LV mass) — primary-prevention ICD indication

5. Follow-up

HCM specialist clinic within 1-2 weeks; cascade family screening (genetic counseling); mavacamten REMS echo q4w during titration then q12w maintenance; ICD/septal reduction decision finalised; sports clearance shared decision; cardiac rehab if NYHA II-III

6. Sources

Guideline: ACC/AHA 2024 HCM Guideline + ESC 2023 cardiomyopathies (Arbelo) + ESC 2014 HCM (Elliott) for SCD risk

  1. pubmed.ncbi.nlm.nih.gov/37622666
  2. pubmed.ncbi.nlm.nih.gov/25173338
  3. pubmed.ncbi.nlm.nih.gov/32861276