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Patient handout

Cardiogenic shock — Long-QT torsades de pointes (TdP) electrical storm

PRODUCTION

1. Your condition

This handout is for cardiogenic shock — long-qt torsades de pointes (tdp) electrical storm. Your care team identified this based on: ecg qtc > 500 ms (bazett or fridericia) with tdp runs on telemetry / 12-lead — tdp electrical storm.

Other reasons your team may use this plan: recurrent syncope or aborted scd in patient with qtc > 480 ms — concern for congenital or acquired lqt with tdp; recent exposure to qt-prolonging drug (class iii antiarrhythmic, macrolide, fluoroquinolone, antipsychotic, methadone, ondansetron, tca, citalopram) precipitating tdp — acquired lqt; family history of sudden death <40 y + qtc > 470 ms (m) or > 480 ms (f) — congenital lqt high concern.

2. Your medications

Take these medications exactly as prescribed. Do not stop or change a dose without talking to your provider.

MedicationStarting doseHowWhenWhat it does
magnesium sulfate2 g IV bolus over 5–15 min then 2 g/h infusion REGARDLESS of measured Mg levelIVcontinuous; titrate to TdP suppressionAHA 2020 ACLS — FIRST-LINE for TdP regardless of measured Mg level; mechanism stabilizes myocardial membrane independent of measured Mg; HRS 2017 PMID 28219760
isoproterenol0.5–2 µg/min IV titrate to HR 90–110IVcontinuous; titrate to HR target + TdP suppressionAHA 2020 ACLS — increases HR which shortens QT and prevents R-on-T; useful for bradycardia-dependent TdP (LQT3 + acquired); CONTRAINDICATED in LQT1/2 acute (adrenergic trigger)
potassium chloride20–40 mEq IV/PO until K ≥4.5IV/POPRNHypokalemia is major TdP precipitant; aggressive K repletion to ≥4.5 mandatory; HRS 2017
norepinephrine0.05–0.5 µg/kg/min IV titrateIVcontinuous; titrate to MAP ≥65SOAP-II PMID 20200382 — first-line in CS; α-1 effect supports MAP; CAUTION in LQT1/2 (adrenergic trigger — minimize dose)
propranololCONGENITAL LQT long-term: propranolol 2–4 mg/kg/d divided BID-QID; do NOT initiate acutely if bradycardia-dependent acquired TdPPOBID-QID; lifelong in congenital LQT1/2Schwartz International LQTS Registry — propranolol mortality reduction in congenital LQT1/2; FIRST-LINE long-term per HRS 2017 Class I
nadololCONGENITAL LQT long-term: nadolol 1–1.5 mg/kg/d dailyPOdaily; lifelong in congenital LQT1/2 (preferred over propranolol per recent registry data)Long half-life + non-selective; recent registry data suggest superior to propranolol in LQT1/2; HRS 2017 Class I

Plan: LQT-TdP storm with CS — MgSO4 + electrolyte repletion + drug withdrawal + pacing/isoproterenol if bradycardia-dependent; AVOID Class IA + III antiarrhythmics; congenital LQT long-term β-blocker

3. When to call your provider

Contact your care team if any of the following happen:

  • Recurrent syncope on β-blocker → emergent EP + ICD / LCSD evaluation
  • New ICD shock → emergent EP + storm investigation
  • New QT-prolonging drug exposure → ED + drug withdrawal + reassessment
  • Family member positive screening → cascade testing + EP referral

4. When to seek emergency care

Call 911 or go to the nearest emergency room right away if you have:

  • Active TdP storm with ongoing QT-prolonging drug exposure — STOP ALL offending drugs immediately (www.crediblemeds.org curated list); MgSO4 2 g IV bolus + 2 g/h infusion; aggressive K + Mg repletion(life-threatening)
  • Refractory TdP despite MgSO4 + isoproterenol + electrolyte repletion + drug withdrawal — temporary transvenous pacing at 80–100 bpm; reconsider diagnosis (myocarditis, structural disease); MCS bridge if SCAI D-E(life-threatening)
  • Bradycardia-dependent TdP (LQT3 or acquired with high-grade AV block / sinus arrest / post-cardioversion pause) — isoproterenol 0.5–2 µg/min OR overdrive pacing at 80–100 bpm; AVOID β-blockers acutely(life-threatening)
  • Distinguish CONGENITAL LQT (family history SCD <40 y, prior syncope, T-wave morphology suggestive) vs ACQUIRED LQT (drug exposure, electrolyte, AKI, bradycardia) — pivots long-term therapy: CONGENITAL → β-blocker FIRST-LINE long-term + cascade testing of relatives + ICD if high-risk + LCSD for refractory; ACQUIRED → drug-avoidance + root-cause + typically QT normalizes within 24–72 h of drug withdrawal

5. Follow-up

CONGENITAL LQT — EP / inherited-arrhythmia clinic 1–4 wks; long-term β-blocker (propranolol or nadolol — Schwartz registry mortality reduction); ICD if high-risk; LCSD for refractory; lifestyle (LQT1 avoid swimming; LQT2 avoid loud noises; LQT3 caution sleep alone); cascade testing first-degree relatives. ACQUIRED LQT — drug-avoidance education + medic-alert bracelet; review home med list against www.crediblemeds.org; root-cause analysis; cardiology follow-up at 1–4 wks; baseline ECG to confirm QTc normalization (most acquired QT prolongation reverses within 24–72 h of drug withdrawal)

6. Sources

Guideline: HRS 2017 Inherited Arrhythmia Syndromes Expert Consensus (Al-Khatib PMID 28219760); AHA 2020 ACLS; Schwartz International LQTS Registry; ESC 2022 VA / SCD prevention; SCAI 2022 CS staging (Naidu PMID 35718438); CredibleMeds (www.crediblemeds.org) curated QT-prolonging drug list

  1. pubmed.ncbi.nlm.nih.gov/28219760
  2. pubmed.ncbi.nlm.nih.gov/33098585
  3. pubmed.ncbi.nlm.nih.gov/35718438