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Patient handout

Cardiogenic shock — fulminant viral / immune myocarditis

PRODUCTION

1. Your condition

This handout is for cardiogenic shock — fulminant viral / immune myocarditis. Your care team identified this based on: recent viral prodrome (uri / gi illness in past 1–4 wks) followed by acute heart failure + cardiogenic shock physiology — fulminant myocarditis with cs.

Other reasons your team may use this plan: cardiac mri: t2 edema + lge (non-ischemic pattern, sub-epicardial / mid-myocardial) + abnormal native t1/t2 mapping (≥2 of 3 = lake louise 2018) + biventricular dysfunction + shock physiology; recent ici exposure (anti-pd1/l1, anti-ctla4) within past 12 weeks + acute hf + ↑ troponin → ici myocarditis, often fulminant; markedly elevated troponin + acute biventricular dysfunction + no obstructive cad on cath → myocarditis differential.

2. Your medications

Take these medications exactly as prescribed. Do not stop or change a dose without talking to your provider.

MedicationStarting doseHowWhenWhat it does
norepinephrine0.05–0.5 µg/kg/min titrate MAP ≥65IVcontinuousSOAP-II PMID 20200382 — NE first-line in CS
dobutamine2.5 µg/kg/min CAUTIOUS titration; AVOID in ventricular electrical stormIVcontinuousDOREMI PMID 33704937 — non-inferior to milrinone; CAUTION in inflamed myocardium given high arrhythmia risk; escalate to MCS rather than higher inotrope doses
methylprednisolone1000 mg IV daily × 3–5 days, then 1 mg/kg/d taperIVdailyAHA 2024 ICI cardiotoxicity statement — high-dose pulse steroids for ICI myocarditis; ESC 2013 myocarditis position paper for eosinophilic / giant cell
cyclosporine3–5 mg/kg/d divided BID; trough target 200–300 ng/mLPO/IVBIDCooper Multicenter Giant Cell Myocarditis Registry NEJM 1997 — combination cyclosporine + steroids markedly improves transplant-free survival vs no immunosuppression (~70% vs ~10%); ESC 2013 myocarditis position paper
abatacept500–1000 mg IV q2 wks × multiple doses per refractory ICI myocarditis protocolIVq2 weeksSalem JACC 2018 + AHA 2024 ICI cardiotoxicity statement — abatacept for steroid-refractory ICI myocarditis; CTLA4-Ig blunts T-cell activation
infliximab5 mg/kg IVIVsingle dose, may repeatAHA 2024 ICI cardiotoxicity statement — alternative to abatacept; CONTRAINDICATED if EF <35 (TNF inhibitor worsens HF per ATTACH NEJM 2003)
phenylephrine40–360 µg/min IVIVcontinuousPure α-pressor; alternative when β-stimulation aggravates arrhythmias in inflamed myocardium
amiodarone150 mg IV bolus then 1 mg/min × 6 h then 0.5 mg/minIVcontinuousAHA 2020 ACLS Class IIb for refractory VT/VF; high arrhythmia risk in inflamed myocardium
metforminAVOID NSAIDsn/an/aAVOID NSAIDs — animal models worsen myocarditis (ESC 2013 position paper); listed as a contraindication marker rather than a drug

Plan: Fulminant myocarditis CS — supportive + cautious inotrope (high arrhythmia risk) + early MCS bridge to recovery + sub-etiology-specific immunosuppression

3. When to call your provider

Contact your care team if any of the following happen:

  • Sustained VT / syncope → EP urgent consult; consider catheter ablation; ICD
  • Persistent severe LV dysfunction → transplant evaluation
  • Recurrent myocarditis → repeat workup; reconsider underlying etiology

4. When to seek emergency care

Call 911 or go to the nearest emergency room right away if you have:

  • Endomyocardial biopsy shows giant cell myocarditis — near-100% mortality without immunosuppression; combination cyclosporine + steroids markedly improves transplant-free survival per Cooper NEJM 1997(life-threatening)
  • Fulminant ICI myocarditis with sustained VT / VF or high-grade AV block — mortality 25–50%; emergency methylprednisolone 1g IV daily + abatacept; permanent ICI cessation(life-threatening)
  • Sustained VT/VF or recurrent shocks within 24 h in fulminant myocarditis — high arrhythmia risk in inflamed myocardium; amiodarone + escalate to MCS over higher inotropes; consider catheter ablation if refractory(life-threatening)
  • Refractory CS in fulminant viral myocarditis — escalate to MCS (IABP / Impella / VA-ECMO) early; recovery is the rule if patient survives initial period (McCarthy NEJM 2000 PMID 10717012); ELSO 2020 ECMO myocarditis registry — 60–70% survival to discharge(life-threatening)
  • New high-grade AV block (Mobitz II / complete heart block) in acute myocarditis — giant cell myocarditis clue (~50%); emergent endomyocardial biopsy + transvenous pacemaker

5. Follow-up

Repeat echo + CMR at 4–8 wks for recovery trajectory; cardiac rehab; the four foundational heart-failure medications 4-pillar if persistent HFrEF; EP follow-up for ICD eligibility per AHA 2017 VA/SCD guideline (waiting period before ICD given recovery potential); psych / oncology follow-up; ICI rechallenge generally CONTRAINDICATED after fulminant ICI myocarditis

6. Sources

Guideline: Caforio ESC 2013 myocarditis position paper (PMID 23824828); Tschöpe AHA 2020 myocarditis scientific statement (PMID 32200645); AHA 2024 ICI cardiotoxicity scientific statement; Cooper Multicenter Giant Cell Myocarditis Registry NEJM 1997; McCarthy NEJM 2000 fulminant vs non-fulminant outcomes (PMID 10717012); Ferreira JACC 2018 Lake Louise Criteria 2018 (PMID 30025572)

  1. pubmed.ncbi.nlm.nih.gov/23824828
  2. pubmed.ncbi.nlm.nih.gov/32200645
  3. pubmed.ncbi.nlm.nih.gov/10717012