← Back to dossier
Patient handout

Cocaine / sympathomimetic hypertensive crisis (alpha-driven HTN + tachycardia + coronary vasospasm)

PRODUCTION

1. Your condition

This handout is for cocaine / sympathomimetic hypertensive crisis (alpha-driven htn + tachycardia + coronary vasospasm). Your care team identified this based on: cocaine, methamphetamine, mdma, or sympathomimetic use within 24-72 h + sbp ≥180 / dbp ≥120 (aha 2008 pmid 18391116; hollander nejm 2008 pmid 18172180).

Other reasons your team may use this plan: hypertensive crisis + tachycardia + diaphoresis + agitation + dilated pupils + chest pain — sympathomimetic toxidrome; ecg ischemia/stemi pattern in setting of acute cocaine/stimulant use — cocaine-associated acs pathway.

2. Your medications

Take these medications exactly as prescribed. Do not stop or change a dose without talking to your provider.

MedicationStarting doseHowWhenWhat it does
lorazepam1-2 mg IV q5-10 min PRN, max 8 mg in 1 hIVPRNAHA 2008 Class I PMID 18391116 — benzodiazepine first for sympatholysis (reduces HTN + tachy + agitation); diazepam alternative; titrate to symptom control
diazepam5-10 mg IV q5-10 minIVPRNAHA 2008 — alternative to lorazepam; longer half-life advantageous for sustained sympatholysis
nitroglycerin5-200 mcg/min IV titrate q3-5 minIVcontinuousAHA 2008 Class IIa — vasodilator + coronary vasodilator; reverses cocaine coronary vasoconstriction; preferred over β-blocker
phentolamine1-5 mg IV q5-15 min titrateIVPRN bolusBoehrer Am J Med 1993 PMID 8390052 — pure alpha-blocker reverses cocaine alpha-vasoconstriction + reverses coronary vasospasm; AHA 2008 Class IIa
verapamil5-10 mg IV slow push q15-30 min OR 0.075-0.15 mg/kgIVPRN bolusNegus Circulation 1994 — verapamil reverses cocaine coronary vasospasm; AHA 2008 Class IIa CCB acceptable
diltiazem0.25 mg/kg IV bolus then 5-15 mg/h infusionIVcontinuousAHA 2008 — alternative non-DHP CCB; useful if AF + RVR
labetalolAVOID as first-line; if used: 10-20 mg IV q10 minIVbolusAHA 2008 — labetalol is debated; mixed α/β theoretically OK but small case reports of paradoxical HTN; AVOID as first-line; reasonable as add-on if benzo + NTG + phentolamine inadequate
AVOID metoprolol/esmolol/propranololAVOIDN/AN/ALange NEJM 1989 PMID 2522592 — propranolol potentiates cocaine coronary vasoconstriction (unopposed alpha); AHA 2008 Class III; ACC/AHA 2025 ACS Class III β-blocker monotherapy in cocaine ACS
AVOID succinylcholine if rhabdomyolysisAVOIDN/AN/AHyperkalemia from rhabdomyolysis + succinylcholine = arrest risk; use rocuronium for RSI

Plan: Cocaine / sympathomimetic HTN crisis — benzodiazepine-first sympatholysis; AVOID β-blocker monotherapy (unopposed alpha); phentolamine reverses alpha-vasoconstriction

3. When to call your provider

Contact your care team if any of the following happen:

  • Relapse with chest pain → ED
  • BP rebound → urgent visit

4. When to seek emergency care

Call 911 or go to the nearest emergency room right away if you have:

  • Cocaine/stimulant use within 24-72 h + STEMI on ECG OR positive troponin trajectory — cocaine-associated MI (rate ~6% per Hollander NEJM 2008)(life-threatening)
  • Core temperature >40 C in cocaine/stimulant toxidrome — risk of multi-organ failure + DIC + death(life-threatening)
  • Cocaine + acute severe back/chest pain + BP differential between arms or pulse deficit — cocaine raises aortic dissection risk 4-5× (Hsue Circulation 2002)(life-threatening)
  • Cocaine/stimulant + sudden severe headache OR new focal deficit OR seizure — cocaine ICH risk (cocaine + HTN + arteritis-like vasculopathy)(life-threatening)
  • Wide-complex tachycardia / brugada-pattern in cocaine toxidrome — sodium-channel blockade by cocaine (TCA-like)(life-threatening)
  • Cocaine + new severe LV apical ballooning + chest pain + minimal CAD on cath — Takotsubo (stress cardiomyopathy) precipitated by cocaine catecholamine surge

5. Follow-up

Substance use disorder counseling + treatment (matrix model for stimulant, contingency management evidence-based per Stitzer 2010); cardiology follow-up if MI occurred (high-risk for recurrent cocaine-MI); echo if Takotsubo suspected (cocaine is precipitant); BP regimen if persistent HTN; opioid agonist therapy if fentanyl-laced supply suspected; harm reduction

6. Sources

Guideline: AHA 2008 Cocaine Cardiovascular Complications Scientific Statement (McCord PMID 18391116) + 2025 ACC/AHA ACS Guideline (Class III β-blocker monotherapy) + 2025 ACC/AHA HTN (Whelton)

  1. pubmed.ncbi.nlm.nih.gov/18391116
  2. pubmed.ncbi.nlm.nih.gov/18172180
  3. pubmed.ncbi.nlm.nih.gov/2522592