← Back to dossier
Patient handout

Pituitary apoplexy

PRODUCTION

1. Your condition

This handout is for pituitary apoplexy. Your care team identified this based on: sudden severe (thunderclap) headache + visual disturbance (rajasekaran clin endocrinol 2011).

Other reasons your team may use this plan: hypotension / shock with sudden headache — secondary adrenal insufficiency (rajasekaran clin endocrinol 2011; briet endocr rev 2015); acute ophthalmoplegia / diplopia (cavernous sinus cn iii/iv/vi) (rajasekaran clin endocrinol 2011); sellar/suprasellar mass with hemorrhage on mri/ct (rajasekaran clin endocrinol 2011).

2. Your medications

Take these medications exactly as prescribed. Do not stop or change a dose without talking to your provider.

MedicationStarting doseHowWhenWhat it does
hydrocortisone100 mg IV/IM bolus STAT, then 50 mg IV q6h OR 200 mg/24h continuous infusion (≥2 mg/h)IVq6h or continuous infusionUK 2011 — secondary (ACTH-deficient) adrenal insufficiency is the life-threatening facet; empiric hydrocortisone is the single most urgent step and is mortality-reducing; give before imaging confirmation
dexamethasone4 mg IV (only if hydrocortisone unavailable, or high-dose anti-edema effect desired with significant chiasmal mass effect)IVq6hUK 2011 — alternative glucocorticoid; preserves cortisol-assay window; some use higher-dose dexamethasone for perichiasmal edema, but hydrocortisone is the default replacement steroid

Plan: Pituitary apoplexy acute — empiric steroid FIRST → resuscitate/Na → image + surgical decision → staged hormone replacement (Rajasekaran Clin Endocrinol 2011)

4. When to seek emergency care

Call 911 or go to the nearest emergency room right away if you have:

  • Hypotension/shock in suspected apoplexy = acute secondary (ACTH-deficient) adrenal insufficiency with no hydrocortisone given (Rajasekaran Clin Endocrinol 2011; Briet Endocr Rev 2015)(life-threatening)
  • Acute or progressive visual acuity/field loss (chiasmal compression) (Rajasekaran Clin Endocrinol 2011)(life-threatening)
  • Reduced consciousness or falling GCS in confirmed/suspected apoplexy (Rajasekaran Clin Endocrinol 2011)(life-threatening)
  • Na <125 with neuro symptoms — cortisol-deficient SIADH-like state vs true SIADH vs delayed DI (Rajasekaran Clin Endocrinol 2011)
  • Postpartum patient with headache + hypopituitarism features — apoplexy vs Sheehan overlap (Rajasekaran Clin Endocrinol 2011)

5. Follow-up

Endocrinology re-evaluation of all anterior axes at 4–8 weeks (most need long-term replacement; ~80% deficient ≥1 axis), interval pituitary MRI, ophthalmology field re-assessment, steroid sick-day rules + emergency hydrocortisone kit + MedicAlert, neurosurgical follow-up of residual adenoma (Rajasekaran Clin Endocrinol 2011; Briet Endocr Rev 2015)

6. Sources

Guideline: 2011 UK Pituitary Apoplexy Guideline (Rajasekaran, Clin Endocrinol) + 2016 Endocrine Society Hypopituitarism Guideline (Fleseriu JCEM) + Briet Endocr Rev 2015 review + 2021–2025 updates

  1. pubmed.ncbi.nlm.nih.gov/21044119
  2. pubmed.ncbi.nlm.nih.gov/26414232
  3. pubmed.ncbi.nlm.nih.gov/27736313