← Back to dossier
Patient handout

Infantile epileptic spasms syndrome (West syndrome)

PRODUCTION

1. Your condition

This handout is for infantile epileptic spasms syndrome (west syndrome). Your care team identified this based on: clusters of brief flexor / extensor / mixed spasms in infant 4-7 mo, often on awakening (go aan/cns 2012 pmid 22689735).

Other reasons your team may use this plan: developmental regression or arrest in infant 3 mo - 2 yr — infantile spasms differential (go aan/cns 2012); hypsarrhythmic (chaotic high-voltage interictal) eeg pattern in infant (go aan/cns 2012); hypopigmented macules (tsc) + spasms in infant — tsc-associated infantile spasms (go aan/cns 2012; iciss o'callaghan 2017).

2. Your medications

Take these medications exactly as prescribed. Do not stop or change a dose without talking to your provider.

MedicationStarting doseHowWhenWhat it does
corticotropinACTH (H.P. Acthar Gel) 75-150 U/m²/day IM divided BID × 2 weeks, then taper × 2 weeks. Low-dose 20 U IM daily may be equivalent to high-dose per Go AAN/CNS 2012 (Go AAN/CNS 2012 PMID 22689735)IMBID × 2 wk then taper × 2 wkGo AAN/CNS 2012 PMID 22689735 — ACTH more effective than vigabatrin short-term (excluding TSC); low-dose probably as effective as high-dose; hormonal therapy may be preferred over VGB in cryptogenic to improve developmental outcome (UKISS Lux 2005 PMID 16239177 cryptogenic VABS benefit at 14 mo)
prednisolone40-60 mg/day PO divided BID-QID × 2 weeks, then taper × 2 weeks (UKISS protocol). Some centers use up to 8 mg/kg/day (Go AAN/CNS 2012; Lux UKISS 2005 PMID 16239177)POBID-QID × 2 wk then taper × 2 wkLux UKISS 2005 PMID 16239177 — prednisolone is an effective hormonal alternative to ACTH (similar short-term efficacy in UKISS); cheaper + easier to administer + oral; UKISS used 40 mg/day BID-QID × 2 wk then taper × 2 wk (UKISS Lux 2005 PMID 16239177)
vigabatrin50 mg/kg/day PO divided BID, titrate over 1-2 weeks to 100-150 mg/kg/day (Go AAN/CNS 2012; Lux UKISS 2005)POBIDGo AAN/CNS 2012 — vigabatrin first-line for TSC-associated IES (more effective in TSC than hormonal); used in combination with hormonal per ICISS O'Callaghan 2017 PMID 27838190 for 72% cessation rate (vs 57% hormonal alone); FDA black box for irreversible peripheral visual field loss — mandatory baseline + q3 mo ophthalmology
pyridoxine100 mg IV under continuous EEG monitoring (one-time trial); response within minutes confirms pyridoxine-dependent epilepsy. May be followed by 30 mg/kg/day PO maintenance if positiveIV then POsingle IV trial then daily PO if positiveAES 2016 Glauser PMID 26900382 + Go AAN/CNS 2012 — pyridoxine-dependent epilepsy mimics IES; B6 trial is low-cost / low-risk / high-impact when positive (response within minutes); document EEG burst-suppression before + after for definitive trial

Plan: Infantile epileptic spasms syndrome — TIME-CRITICAL first-line (3 options + combination) (Go AAN/CNS 2012 PMID 22689735; ICISS O'Callaghan 2017 PMID 27838190)

3. When to call your provider

Contact your care team if any of the following happen:

  • Refractory spasms or developmental regression → urgent admit for treatment initiation (Go AAN/CNS 2012)
  • Refractory status epilepticus → ED + route to peds.status_epilepticus.v1 (AES 2016 Glauser PMID 26900382)
  • Suspected TSC → cardiology echo + skin Wood's lamp + brain MRI + dermatology + ophthalmology (Go AAN/CNS 2012)
  • Suspected IEM → metabolism / genetics + IEM workup (Go AAN/CNS 2012)

4. When to seek emergency care

Call 911 or go to the nearest emergency room right away if you have:

  • New diagnosis of infantile epileptic spasms syndrome — TIME-CRITICAL treatment window (Go AAN/CNS 2012 PMID 22689735)
  • IES with TSC features (hypopigmented macules, cardiac rhabdomyomas, brain MRI tubers, family history) (Go AAN/CNS 2012)
  • Persistent spasms after 2 weeks of first-line hormonal OR vigabatrin (Go AAN/CNS 2012)
  • Infant < 18 mo with refractory spasms or diagnostic uncertainty — pyridoxine-dependent epilepsy trial (AES 2016 Glauser PMID 26900382; Go AAN/CNS 2012)
  • Vigabatrin visual field changes on ophthalmology surveillance (FDA black box for irreversible peripheral visual field loss)
  • ACTH or prednisolone serious side effects: uncontrolled HTN, hyperglycemia requiring insulin, severe infection (PJP, varicella, bacterial sepsis), cardiac toxicity (Lux UKISS 2005)
  • Spasm cessation but evolution to Lennox-Gastaut syndrome (multiple seizure types + slow spike-wave on EEG) OR other refractory epilepsy syndrome (Go AAN/CNS 2012)

5. Follow-up

Pediatric neurology / epileptology q1-3 mo; developmental pediatrics; early intervention; genetic counseling if positive; family education on safety / monitoring / triggers (Go AAN/CNS 2012; Lux UKISS 2005)

6. Sources

Guideline: Go AAN/CNS 2012 PMID 22689735 (evidence-based guideline update: medical treatment of infantile spasms) + Lux UKISS 2005 PMID 16239177 (UK Infantile Spasms Study) + Darke 2010 PMID 20457702 (UKISS 4-yr follow-up) + O'Callaghan ICISS 2017 PMID 27838190 (combination hormonal + vigabatrin)

  1. pubmed.ncbi.nlm.nih.gov/22689735
  2. pubmed.ncbi.nlm.nih.gov/16239177
  3. pubmed.ncbi.nlm.nih.gov/20457702