← Back to dossier
Patient handout

Idiopathic pulmonary fibrosis (IPF)

PRODUCTION

1. Your condition

This handout is for idiopathic pulmonary fibrosis (ipf). Your care team identified this based on: progressive dyspnea on exertion (>3–6 mo) (ats/ers 2022).

Other reasons your team may use this plan: persistent dry cough (ats/ers 2022); bibasilar velcro inspiratory crackles ± digital clubbing (ats 2018 dx); hrct uip pattern (basal, subpleural, reticulation, honeycombing, traction bronchiectasis) (ats 2018 dx).

2. Your medications

Take these medications exactly as prescribed. Do not stop or change a dose without talking to your provider.

MedicationStarting doseHowWhenWhat it does
pirfenidone267 mg PO TID wk1, 534 mg TID wk2, then 801 mg TID maintenancePOTID WITH FOODASCEND (King NEJM 2014 PMID 24836312): ≥10% FVC decline-or-death relative reduction ~47.9% at 52 wk; CAPACITY pooled (Noble Lancet 2011 PMID 21571362) confirmatory. Prefer when nintedanib bleeding/anticoagulant risk dominates. Photosensitivity + GI + hepatotoxicity — take with food, sun protection; reduce to 534 then 267 mg TID for ADR
nintedanib150 mg PO BID (consider 100 mg BID if hepatic impairment / poor tolerance)POBID WITH FOODINPULSIS-1/2 (Richeldi NEJM 2014 PMID 24836310): annual FVC −114.7 vs −239.9 mL (Δ ~125 mL/yr); also slows non-IPF PF-ILD (INBUILD PMID 31566307: −80.8 vs −187.8 mL/yr) and SSc-ILD (SENSCIS PMID 31112379). Diarrhoea most common ADR (loperamide + dose-reduce 150→100 mg BID); hepatotoxicity; bleeding + arterial thromboembolic risk — caution on full anticoagulation; CYP3A4/P-gp substrate
oxygentitrate to resting SpO2 ≥90%; ambulatory O2 for exertional desaturationinhaledcontinuous if resting SpO2 ≤88%ATS/ERS 2022 — LTOT for resting hypoxemia (SpO2 ≤88%); ambulatory O2 for isolated exertional desaturation (symptom/QoL benefit)
pulmonary rehabilitationstructured exercise + education programn/aprogram course; maintenance thereafterATS/ERS 2022 — conditional recommendation; improves 6MWD, dyspnea, QoL
lung transplant referralEARLY referral at diagnosis if no absolute contraindicationn/aone-time referral + ongoing co-managementLung transplant is the ONLY disease-modifying option; IPF has worst waitlist mortality among ILDs — refer early (ISHLT candidate-selection principles; ATS/ERS 2022)
omeprazole20–40 mgPOonce dailyAnti-reflux is CONDITIONAL (low-quality evidence; 2022 update softened the 2015 recommendation) — treat symptomatic GERD; routine anti-acid for asymptomatic IPF NOT recommended
esomeprazole20–40 mgPOonce dailyPPI alternative when omeprazole CYP2C19 interactions are problematic; same CONDITIONAL anti-reflux rationale
methylprednisolone500–1000 mg IV daily ×3 (pulse), then taper; OR prednisone 0.5–1 mg/kg/dayIV/POpulse ×3 then taper over weeksAE-IPF in-hospital mortality 50-60% (Collard 2016 PMID 27299520). High-dose corticosteroid is common practice but WEAK/no-RCT evidence (conditional-against in 2022 guideline; individualise + treat precipitant); continue antifibrotic if tolerating PO
prednisone0.5–1 mg/kg/day POPOtaper over weeksOral corticosteroid option for AE-IPF when IV pulse not used. NEVER as part of triple therapy (PANTHER PMID 22607134 — pred+azathioprine+NAC increased death & hospitalisation; STOP if patient on legacy triple)
nintedanib 100 mg BID (hepatic impairment — Child-Pugh A; AVOID in Child-Pugh B/C)100 mg PO BID (reduced)POBID with foodSpecial-pop: hepatic impairment branch — nintedanib Child-Pugh A reduce to 100 mg BID; NOT recommended Child-Pugh B/C (DailyMed label). Pirfenidone also requires caution / dose-reduction in moderate hepatic impairment and is contraindicated in severe — prefer best-tolerated agent with intensified LFT
pirfenidone (anticoagulated patient — preferred over nintedanib)801 mg TID maintenance (standard titration)POTID with foodSpecial-pop: anticoagulated branch — nintedanib carries bleeding + arterial thromboembolic signal and VEGFR inhibition; in patients on full-dose anticoagulation or high bleed risk, prefer PIRFENIDONE. If nintedanib unavoidable, co-manage anticoagulation intensity and monitor closely
antifibrotic peri-transplant managementcontinue antifibrotic to bridge waitlist; coordinate hold timing with transplant surgery (wound-healing/bleeding considerations — nintedanib anti-angiogenic)n/auntil transplant per center protocolSpecial-pop: peri-transplant branch — antifibrotics bridge progression on the waitlist; nintedanib VEGFR inhibition raises perioperative wound-healing/bleeding concern → hold timing is center-specific; do not abruptly stop without transplant-team coordination
AE-IPF supportive bundle (HFNC/NIV, treat precipitant, palliative-aligned ICU decision)O2 to SpO2 ≥90%, HFNC/NIV trial, broad infection workup + empiric coverage, VTE assessmentn/acontinuous during AE-IPFSpecial-pop: AE-IPF branch — invasive ventilation has very high mortality and should generally be reserved for transplant candidates as a bridge; align ICU/ECMO escalation with goals of care and palliative input (Collard 2016 PMID 27299520)
pirfenidone (elderly / comorbid CPFE — start best-tolerated, intensify monitoring)267 mg TID slow titration; individualisePOTID with foodSpecial-pop: elderly / CPFE branch — CPFE (emphysema + lower-lobe fibrosis) has preserved spirometric volumes but severe ↓DLCO and disproportionate PH; antifibrotic still indicated for the fibrotic component. In frail elderly use slow titration, the best-tolerated agent, and tighter LFT/ADR surveillance; screen Group-3 PH (route pulm.pulmonary_htn_group2_to_5.v1)

Plan: IPF antifibrotic + supportive — chronic progressive, ATS/ERS/JRS/ALAT 2022

3. When to call your provider

Contact your care team if any of the following happen:

  • Acute exacerbation (acute dyspnea + new GGO) → ED (Collard 2016 PMID 27299520)
  • Resting hypoxemia oxygen level (SpO₂) ≤88% → LTOT (ATS/ERS 2022)
  • FVC ≥10% relative decline in 6 mo or DLCO ≤35% → expedite transplant evaluation + palliative (Ley 2012)

4. When to seek emergency care

Call 911 or go to the nearest emergency room right away if you have:

  • AE-IPF (Collard 2016 PMID 27299520) — acute worsening dyspnea <30 d + new bilateral GGO/consolidation NOT fully explained by HF, fluid overload, PE or infection(life-threatening)
  • GAP Stage III (8–12 pts) OR rapid progression (FVC ≥10% relative decline in 6 mo OR DLCO ≥15% decline) — 1-yr mortality ~39%
  • Resting oxygen level (SpO₂) ≤88% on room air
  • Patient found on prednisone + azathioprine + N-acetylcysteine triple therapy for IPF
  • Acute deterioration on background IPF — AE-IPF vs mimic? §5.5.2 AE-IPF differential (Collard 2016 PMID 27299520): infection (viral PCR + bacterial + PCP if immunosuppressed) vs PE (CTPA — D-dimer NON-diagnostic in IPF) vs cardiogenic edema (natural marker of fluid overload (BNP)↑↑, echo LV dysfunction) vs aspiration (witnessed/risk factors) vs drug-induced ILD (recent culprit drug) vs idiopathic AE-IPF (diagnosis of exclusion)(life-threatening)

5. Follow-up

ILD clinic q3–6 mo, transplant clinic co-management, comorbidity sweep (Group-3 PH, CPFE, GERD, OSA, lung-cancer surveillance — IPF raises lung-ca risk, depression), pulmonary rehab continuity, palliative care + advance care planning (ATS/ERS 2022)

6. Sources

Guideline: ATS/ERS/JRS/ALAT 2022 IPF (update) + Progressive Pulmonary Fibrosis Guideline (PMID 35486072); ATS/ERS/JRS/ALAT 2018 IPF Diagnosis Guideline (PMID 30168753). Current as of 2026-05-18 — no 2024/25 ATS/ERS/JRS/ALAT IPF replacement (WebSearch-confirmed).

  1. pubmed.ncbi.nlm.nih.gov/35486072
  2. pubmed.ncbi.nlm.nih.gov/30168753
  3. pubmed.ncbi.nlm.nih.gov/24836312