Post-cardiac-arrest care — electrical-injury-induced arrest
Encounter flow
12/12 authoredCanonical 12-phase frame with authored status for this dossier.
Frame
Electrical-injury cardiac arrest — high-voltage / lightning typically asystole; low-voltage AC typically VF via R-on-T; post-ROSC dominated by occult deep-tissue injury (rhabdo + AKI), cardiac contusion, transient conduction abnormalities, and fall-related trauma; route to parent cardio.post-arrest.core.v1 for TTM + neuroprog
electrical etiology + voltage class + pathway documented
Patient inputs (20)
Pediatric vs adult electrical injury patterns differ (oral commissure burns in toddlers; occupational pattern in working-age adults); age also affects rhabdomyolysis tolerance + AKI risk
High-voltage (>1000 V) → asystole + extensive deep-tissue injury + multi-system trauma; low-voltage (<1000 V AC) → VF predominant; lightning → unique pattern (Lichtenberg figures, keraunoparalysis, asystole that often spontaneously reverts)
Hand-to-hand or hand-to-foot pathway = transthoracic current → highest cardiac arrest risk; estimating entry/exit wound location informs the deep-tissue injury workup
Witnessed + bystander CPR + low-flow time → favorable neuro prognosis (AHA 2020); CAHP/OHCA score inputs
High-voltage typically asystole; low-voltage AC typically VF; lightning often asystole that reverts spontaneously then deteriorates to secondary VF/asystole from hypoxia
CPR duration → ECPR eligibility (ARREST PMID 33308475 — <60 min low-flow); neuro prognosis weighting
Electrical shock often produces tetanic muscle contraction → fall from height or blast injury → C-spine + head + long-bone injuries; mandates trauma evaluation
AKI from myoglobinuria + post-arrest hypoperfusion; baseline + serial trending q6-12h drives renal-replacement consideration; KDIGO 2012 staging
Often normal initially after ROSC, but 25-50% develop transient AV blocks / QTc prolongation / ischemic patterns from coronary spasm in first 24h — serial ECGs at 4-6h interval (Spies 2006 PMID 17143257)
Cardiac contusion + coronary spasm both produce troponin elevation; rise pattern helps distinguish electrical-injury myocardial damage from concomitant ACS triggered by stress
CK-total marker for skeletal muscle injury → rhabdomyolysis; CK-MB can be misleading if total CK very high (skeletal contribution); CK >5000 U/L = high AKI risk threshold (Bosch 2009 PMID 19571284)
Myoglobinuria → pigment nephropathy → AKI; dipstick "blood" positive without RBCs on microscopy = myoglobinuria pattern; quantitative serum or urine myoglobin available in many centers
Tissue hypoperfusion + anaerobic metabolism from deep-tissue ischemia + post-arrest perfusion debt; clearance trajectory drives prognosis (SCAI 2022 PMID 35718438)
Hyperkalemia from cell-membrane damage + rhabdo + AKI; potentially massive if severe rhabdo; correct to 4-4.5 with insulin/glucose ± dialysis if refractory
Aspiration / pneumothorax from CPR / inhalation injury / blast pattern; baseline for ARDS evolution
MAP ≥65 target post-ROSC; hypovolemia from third-spacing / burn shock common with high-voltage; SCAI staging if shock
TTM target 33-37.5 °C × 24h (TTM2 PMID 34133859); not contraindicated by burn injury but coordinate with burn center
Avoid hyperoxia: SpO2 92-98% (AHA 2020 Class IIa); inhalation injury possible if arc/blast exposure
Tetanic contraction-related fall → C-spine + head injury; AMS post-ROSC mandates CT head; cervical immobilization until cleared
LV/RV function for cardiac contusion vs post-arrest stunning; baseline for serial follow-up
* = hard-required. Engine cannot meaningfully run until these are filled.
Severity triggers (4)
- informationallife_threateningcompartment_syndrome_post_electrocutionFirm/tense extremity compartment + pain out of proportion + decreased pulses + paresthesia + measured pressure >30 mmHg or delta-P <30 mmHgTrigger could not be auto-evaluated — needs clinician judgement.
- informationallife_threateningsevere_hyperkalemia_above_6_with_rhabdo_akiSerum K >6.0 with ECG changes (peaked T waves, widened QRS) in setting of rhabdo + AKI post-electrocutionTrigger could not be auto-evaluated — needs clinician judgement.
- informationalsevererhabdomyolysis_with_aki_post_electrocutionCK >5000 U/L with rising creatinine and / or myoglobinuria in post-electrocution patient — myoglobinuric AKI (Bosch 2009 PMID 19571284; KDIGO 2012 §5.4)Trigger could not be auto-evaluated — needs clinician judgement.
- informationalseveretransient_av_block_or_qtc_prolongation_post_electrocutionNew AV block (any degree) or QTc >500 ms on serial ECG within first 24h post-electrocution — Spies 2006 PMID 17143257Trigger could not be auto-evaluated — needs clinician judgement.
Workflow calculators
Run this disease's risk and dosing calculators inline.
Recommended regimen
Electrical-injury post-arrest phenotype — IVF rhabdo bundle + cardiac conduction surveillance + post-ROSC standard bundle (AHA 2020 + Spies 2006 + Bosch 2009 + KDIGO 2012)- norepinephrinefirst linevasopressor_alpha1_beta10.05-0.5 µg/kg/min titrate MAP ≥65 • IV • continuoustriggers: post_rosc_vasoplegia, burn_shock_with_distributive_componentSOAP-II PMID 20200382; first-line post-ROSC vasoactiverxcui 7512
- amiodaronefirst lineclass_iii_antiarrhythmic300 mg IV bolus → 150 mg if recurrent → 1 mg/min × 6h → 0.5 mg/min × 18h • IV • standard ACLS dosingtriggers: recurrent_vf_pvt_post_electrocution, sustained_vt_with_pulseAHA 2020 ACLS Class IIb; standard cadence (no hypothermia adjustment unless TTM in deep-cool phase)rxcui 703
- epinephrinefirst lineinotrope_chronotrope_vasopressor1 mg IV q3-5 min during arrest • IV • standard ACLStriggers: cardiac_arrest, pea_asystole_during_index_arrestAHA 2020 ACLSrxcui 3992
- sodium bicarbonateadd onalkalinizing_agent150 mEq in 1 L D5W at 150-250 mL/h to target urine pH ≥6.5 • IV • continuous infusiontriggers: myoglobinuria_with_acidic_urine, severe_metabolic_acidosis_with_rhabdoKDIGO 2012 §5.4 — urinary alkalinization for myoglobinuric AKI prevention; controversial Class IIb but commonly used; monitor pH + bicarb + Carxcui 8591
- calcium gluconatefirst lineelectrolyte_membrane_stabilizer1-3 g IV over 5-10 min • IV • as needed for hyperkalemia ECG changestriggers: hyperkalemia_with_ecg_changes, severe_hyperkalemia_above_6_5KDIGO 2020 hyperkalemia — membrane stabilization within minutes; precedes shifting therapiesrxcui 1908
- insulin regularfirst lineshort_acting_insulin10 units IV with 25 g D50 • IV • as needed for hyperkalemiatriggers: hyperkalemia_above_6_0_persistent_after_calciumKDIGO 2020 hyperkalemia — intracellular K shift; monitor glucose q1h × 4-6hrxcui 253182
- magnesium sulfateadd onelectrolyte_anti_arrhythmic1-2 g IV • IV • one-time + repeat for TdPtriggers: torsades_de_pointes_post_electrocution_qtc_prolongation, long_qt_unmaskedAHA 2020 ACLS Class IIa for TdP; QTc prolongation common in first 24h post-electrocutionrxcui 6585
- propofolfirst linesedative_iv_anesthetic5-50 µg/kg/min • IV • continuous; titrate RASStriggers: post_rosc_intubation_ttmPADIS 2018rxcui 8782
- fentanylfirst lineopioid_analgesic25-200 µg/h • IV • continuoustriggers: post_rosc_intubation_ttm, burn_pain_management, shivering_controlPADIS 2018; analgesia + shivering suppression for TTMrxcui 4337
- tranexamic acidadd onantifibrinolytic1 g IV over 10 min • IV • within 3h of bleeding onsettriggers: concomitant_traumatic_bleeding_from_fall, hemorrhagic_shock_co_existingCRASH-2 PMID 20554319 if traumatic bleeding co-existing with electrocutionrxcui 10691
outpatient playbook — drug actions (2)
- 1. continue channelopathy regimen if unmaskedrxcui 6918nadolol or metoprolol per indication • PO • dailytrigger: channelopathy or persistent QTc/arrhythmiaHRS 2017
- 2. continue HF GDMT if structural CMrxcui 1656328sacubitril-valsartan + carvedilol + spironolactone + SGLT2i max tolerated • PO • as scheduledtrigger: structural CM HFrEFACC/AHA 2022 HF 4-pillar
Auto-drafted A&P note
outpatientSubjective
- Possible entry pathways: ROSC after electrical-injury cardiac arrest (high-voltage industrial, lightning, or low-voltage household AC); High-voltage (>1000 V) industrial exposure or lightning strike with cardiac arrest — asystole most common (AHA 2020 §electrical injury); Low-voltage household AC (110/220 V, 50-60 Hz) with VF — R-on-T mechanism during vulnerable T-wave window (Spies 2006 PMID 17143257).
Objective
- No vitals, labs, or imaging entered for this encounter.
Assessment
**Post-cardiac-arrest care — electrical-injury-induced arrest** (cardio.post-arrest.electrocution.v1). Phenotype framing: Primary electrical-injury arrest (direct cardiac depolarisation) vs secondary (hypoxia from prolonged tetany, fall-trauma) vs unmasked underlying disease (LQTS, Brugada, ischemic) vs concomitant injury (TBI from fall, hemorrhagic shock) Scope: Electrical-injury cardiac arrest — high-voltage / lightning typically asystole; low-voltage AC typically VF via R-on-T; post-ROSC dominated by occult deep-tissue injury (rhabdo + AKI), cardiac contusion, transient conduction abnormalities, and fall-related trauma; route to parent cardio.post-arrest.core.v1 for TTM + neuroprog No severity triggers fired against current inputs.
Plan
Regimen axis: **Electrical-injury post-arrest phenotype — IVF rhabdo bundle + cardiac conduction surveillance + post-ROSC standard bundle (AHA 2020 + Spies 2006 + Bosch 2009 + KDIGO 2012)**. 1. norepinephrine 0.05-0.5 µg/kg/min titrate MAP ≥65 IV continuous (vasopressor_alpha1_beta1, first line) — SOAP-II PMID 20200382; first-line post-ROSC vasoactive 2. amiodarone 300 mg IV bolus → 150 mg if recurrent → 1 mg/min × 6h → 0.5 mg/min × 18h IV standard ACLS dosing (class_iii_antiarrhythmic, first line) — AHA 2020 ACLS Class IIb; standard cadence (no hypothermia adjustment unless TTM in deep-cool phase) 3. epinephrine 1 mg IV q3-5 min during arrest IV standard ACLS (inotrope_chronotrope_vasopressor, first line) — AHA 2020 ACLS 4. sodium bicarbonate 150 mEq in 1 L D5W at 150-250 mL/h to target urine pH ≥6.5 IV continuous infusion (alkalinizing_agent, add on) — KDIGO 2012 §5.4 — urinary alkalinization for myoglobinuric AKI prevention; controversial Class IIb but commonly used; monitor pH + bicarb + Ca 5. calcium gluconate 1-3 g IV over 5-10 min IV as needed for hyperkalemia ECG changes (electrolyte_membrane_stabilizer, first line) — KDIGO 2020 hyperkalemia — membrane stabilization within minutes; precedes shifting therapies 6. insulin regular 10 units IV with 25 g D50 IV as needed for hyperkalemia (short_acting_insulin, first line) — KDIGO 2020 hyperkalemia — intracellular K shift; monitor glucose q1h × 4-6h 7. magnesium sulfate 1-2 g IV IV one-time + repeat for TdP (electrolyte_anti_arrhythmic, add on) — AHA 2020 ACLS Class IIa for TdP; QTc prolongation common in first 24h post-electrocution 8. propofol 5-50 µg/kg/min IV continuous; titrate RASS (sedative_iv_anesthetic, first line) — PADIS 2018 9. fentanyl 25-200 µg/h IV continuous (opioid_analgesic, first line) — PADIS 2018; analgesia + shivering suppression for TTM 10. tranexamic acid 1 g IV over 10 min IV within 3h of bleeding onset (antifibrinolytic, add on) — CRASH-2 PMID 20554319 if traumatic bleeding co-existing with electrocution Setting playbook (outpatient) — Long-term cardiology / EP / nephrology surveillance; ICD management if channelopathy or structural CM; mental health long-term; occupational medicine if work-related 11. continue channelopathy regimen if unmasked nadolol or metoprolol per indication PO daily — channelopathy or persistent QTc/arrhythmia (HRS 2017) 12. continue HF GDMT if structural CM sacubitril-valsartan + carvedilol + spironolactone + SGLT2i max tolerated PO as scheduled — structural CM HFrEF (ACC/AHA 2022 HF 4-pillar) Non-pharmacologic actions: - ICD/WCD adherence - Workplace safety + PPE long-term - Family CPR/AED ongoing - Mental health long-term AVOID / contraindication checks: - Beta_blocker_avoid_acute_cardiogenic_shock (AHA 2020) - Succinylcholine_avoid_severe_rhabdo_with_hyperkalemia (anesthesia consensus) - Nephrotoxic_drug_avoid_evolving_aki (KDIGO 2012) - Nsaid_avoid_aki_or_rhabdo (KDIGO 2012)
Monitoring
Regimen monitoring: - continuous ecg telemetry x 24h for transient conduction block (AHA 2020 + Spies 2006) - serial ecg q4-6h x 24h for qtc av block evolution (Spies 2006 PMID 17143257) - serial troponin q3-6h x 24h (4th UDMI 2018) - CK q6h until peak then q12h until downtrending (Bosch 2009 PMID 19571284) - BMP q6h + K + Mg + Ca + bicarb (rhabdo electrolyte derangement) - urine output hourly target 1-2 mL/kg/h (KDIGO 2012 §5.4) - urine pH q2-4h if alkalinizing target pH above 6.5 (KDIGO 2012) - compartment exam q4h x 24h then q8h (orthopedic literature) - continuous core temp via bladder or esophageal probe during TTM (TTM2) - continuous EEG for 24-48h (Sandroni 2021) - NSE at 24h 48h 72h (Sandroni 2021) - lactate q2-4h until normalized (SCAI 2022) Setting (outpatient) monitoring: - Quarterly BP + weight + symptom score - Annual ECG + Holter if channelopathy - Annual echo if HFrEF Follow-up plan: Cardiology follow-up at 1-2 weeks for QTc + LV function reassessment; nephrology if AKI persists; PT/OT for deep-tissue + burn rehab; trauma surgery for fracture / spinal injuries; mental health (PTSD risk very high); occupational medicine + workers-comp documentation if work-related; family CPR/AED training; cardiac MRI if suspected channelopathy unmasked or persistent dysfunction - Close-out criterion: cardiology + nephrology + rehab + mental health + occupational follow-up booked Monitoring phase: Continuous telemetry (capture transient AV blocks for first 24h) + arterial line + central line + Foley with hourly UOP target 1-2 mL/kg/h; CK q6h until peak documented + downtrending; BMP q6h with K + Cr + bicarb; serial troponin; serial neuro exams; multimodal neuroprog ≥72h post-rewarm (Sandroni 2021 PMID 33745427); compartment exam q4h × 24h then q8h
Disposition
Current setting: outpatient — Long-term cardiology / EP / nephrology surveillance; ICD management if channelopathy or structural CM; mental health long-term; occupational medicine if work-related Disposition criteria: - Long-term continuation; cross-link to cardio.hf.core.v1 for chronic HFrEF Escalation triggers (move to higher acuity): - ICD therapy delivered → urgent EP; consider catheter ablation per VANISH (PMID 27149033) if structural substrate - EF declining despite GDMT → advanced HF eval
Earlier-Return Triggers
Return-precaution thresholds (watch for): - [LIFE_THREATENING] Firm/tense extremity compartment + pain out of proportion + decreased pulses + paresthesia + measured pressure >30 mmHg or delta-P <30 mmHg - [LIFE_THREATENING] Serum K >6.0 with ECG changes (peaked T waves, widened QRS) in setting of rhabdo + AKI post-electrocution - [SEVERE] CK >5000 U/L with rising creatinine and / or myoglobinuria in post-electrocution patient — myoglobinuric AKI (Bosch 2009 PMID 19571284; KDIGO 2012 §5.4)
Citations
- AHA 2020 ACLS / Post-Cardiac-Arrest Care electrical injury § + Spies 2006 narrative review + Koumbourlis 2002 + KDIGO 2012 AKI + KDIGO 2020 hyperkalemia + TTM2 + HYPERION + Sandroni 2021 neuroprog [PMID:33081530](https://pubmed.ncbi.nlm.nih.gov/33081530/) - Cited evidence (PMID 17143257) [PMID:17143257](https://pubmed.ncbi.nlm.nih.gov/17143257/) - Cited evidence (PMID 12528783) [PMID:12528783](https://pubmed.ncbi.nlm.nih.gov/12528783/) - Cited evidence (PMID 19571284) [PMID:19571284](https://pubmed.ncbi.nlm.nih.gov/19571284/) - Cited evidence (PMID 34133859) [PMID:34133859](https://pubmed.ncbi.nlm.nih.gov/34133859/) Last reconciled with current guidelines: 2026-05-15.
- AHA 2020 ACLS / Post-Cardiac-Arrest Care electrical injury § + Spies 2006 narrative review + Koumbourlis 2002 + KDIGO 2012 AKI + KDIGO 2020 hyperkalemia + TTM2 + HYPERION + Sandroni 2021 neuroprog — PMID:33081530
- Cited evidence (PMID 17143257) — PMID:17143257
- Cited evidence (PMID 12528783) — PMID:12528783
- Cited evidence (PMID 19571284) — PMID:19571284
- Cited evidence (PMID 34133859) — PMID:34133859